Can hypoglycemia cause cerebral palsy?
Persistent or recurrent hypoglycemia can result in neonatal permanent brain injury, leaving cognitive impairment, vision disturbance, occipital lobe epilepsy, cerebral palsy and other sequelae.
Can hyperglycemia cause cerebral palsy?
As dissimilar as they may seem, there is a connection. Research tells us that gestational diabetes (diabetes that occurs during pregnancy) can put a baby at risk of being born with cerebral palsy.
Why does hypoglycemia cause neurological symptoms?
These symptoms prompt individuals to ingest food to increase blood sugar levels. If these defenses are unable to restore blood sugar levels, inadequate glucose supply to the brain leads to neuroglycopenic symptoms such as confusion, difficulty speaking, ataxia, paresthesias, headaches, seizures, and coma.
What are differential diagnosis for cerebral palsy?
Cerebral palsy must be differentiated from other diseases that cause spasticity, hypotonia, ataxia, and dystonia such as inherited metabolic disorders, intellectual disability, metabolic myopathies, metabolic neuropathy, traumatic peripheral nerve lesions, tumors of the conus and cauda equina and vascular malformations …
Can low blood sugar lead to diabetes?
If you have diabetes, episodes of low blood sugar are uncomfortable and can be frightening. Fear of hypoglycemia can cause you to take less insulin to ensure that your blood sugar level doesn’t go too low. This can lead to uncontrolled diabetes.
Does diabetes cause palsy?
Research in the adult population with diabetes shows a correlation between poor glycemic control and the development of Bell palsy. The more elevated the glycated hemoglobin level, the more severe the facial nerve palsy.
What are the neurological symptoms of hypoglycemia?
Neuroglycopenic symptoms include weakness, tiredness, or dizziness; inappropriate behavior (sometimes mistaken for inebriation), difficulty with concentration; confusion; blurred vision; and, in extreme cases, coma and death.
What causes spastic cerebral palsy?
Spastic cerebral palsy is caused by damage to the motor cortex and the pyramidal tracts of the brain, which connect the motor cortex to the spinal cord. Understanding the function of the motor cortex and pyramidal tracts helps to explain how damage to these systems affects movement in those with spastic CP.