Is milk thistle good for cirrhosis of the liver?
In a comprehensive review of studies on milk thistle by the U.S. Agency for Healthcare Research and Quality (AHRQ), milk thistle improved liver function in people with mild liver disease, but was less effective for those with severe liver disease, such as cirrhosis.
Is silymarin good for cirrhosis?
Clinically, silymarin reduces liver dysfunction, may reduce liver-related mortality in patients with cirrhosis and improves glycemic control in patients with concomitant diabetes, with few if any adverse events.
What vitamins should I take with cirrhosis?
I also recommend that patients with cirrhosis take a multivitamin, and in some patients, supplementation is recommended for select micronutrients such as zinc. The standard daily dose of zinc is 50 mg/day of elemental zinc, which is the amount of zinc in a 220-mg zinc sulfate tablet.
Can you get alcoholic hepatitis if you drink alcohol?
However, the relationship between drinking and alcoholic hepatitis is complex. Not all heavy drinkers develop alcoholic hepatitis, and the disease can occur in people who drink only moderately. If you’re diagnosed with alcoholic hepatitis, you must stop drinking alcohol.
What are the signs and symptoms of alcoholic hepatitis?
Additional signs and symptoms that occur with severe alcoholic hepatitis include: 1 Fluid accumulation in your abdomen (ascites). 2 Confusion and behavior changes due to a buildup of toxins normally broken down and eliminated by the liver. 3 Kidney and liver failure.
How is milk thistle used in the treatment of hepatitis?
Despite the fact that milk thistle is widely used in the treatment of hepatitis (particularly hepatitis C), results from four viral hepatitis studies were contradictory. Some found improvements in liver enzyme activity while others failed to detect these benefits.
What is the differential diagnosis of alcoholic hepatitis?
The differential diagnosis of alcoholic hepatitis includes nonalcoholic steatohepatitis, acute or chronic viral hepatitis, drug-induced liver injury, fulminant Wilson’s disease, autoimmune liver disease, alpha-1 antitrypsin deficiency, pyogenic hepatic abscess, ascending cholangitis, and decompensation associated with hepatocellular carcinoma.